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Book cover for Oxford Handbook of Expedition and Wilderness Medicine (2 edn) Oxford Handbook of Expedition and Wilderness Medicine (2 edn)
Chris Johnson et al.

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Contents

July 26, 2018: This chapter has been re-evaluated and remains up-to-date. No changes have been necessary

Disclaimer
Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct. Readers must therefore always … More Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct. Readers must therefore always check the product information and clinical procedures with the most up to date published product information and data sheets provided by the manufacturers and the most recent codes of conduct and safety regulations. The authors and the publishers do not accept responsibility or legal liability for any errors in the text or for the misuse or misapplication of material in this work. Except where otherwise stated, drug dosages and recommendations are for the non-pregnant adult who is not breastfeeding.

Limb injuries may be life-threatening and an initial ABC evaluation should be performed, with respiratory and circulation status regularly re-evaluated (see graphic Initial response to an incident, p. 180; graphic Assessment of a casualty, p. 185). Consider the possibility of associated head, spine, chest, abdominal, or pelvic injuries, particularly if the patient is unconscious.

Approach the casualty if safe to do so.

Ensure that the airway is open, assess the breathing rate, and look for signs of shock (graphic Shock, p. 228). Remember the possibility of cervical spine injury with any significant injury above the collarbones, with multiple injuries, and with head injuries resulting in unconsciousness.

Control any external bleeding with direct pressure. If the bleeding is catastrophic (traumatic amputation), apply a tourniquet (graphic Control of haemorrhage, p. 196).

Anticipate and treat shock, particularly with thigh fractures. Where appropriate use a suitable traction splint.

Remember that painful limb injuries may distract attention from less painful but more significant trunk injuries.

Expose the affected part and cut off clothing only if absolutely necessary given that in some circumstances clothing, footwear, and waterproofs will be needed to protect the patient from the environment.

Look for signs of an interrupted blood supply—pale, pulseless, painful, perishingly cold.

With fractures/dislocations, attempt manipulation early in an attempt to restore distal circulation. Check pulses and sensation before and after any manipulation.

Look for nerve damage affecting movement and/or sensation.

Give painkillers.

Splint fractures using commercially available or improvised materials.

Transfer to suitable shelter.

Sleeping mat.

Paddles.

Skis or ski poles.

Slings and karabiners.

Tree branches.

Sleeping bags.

Ropes.

When applying splints, remember that they must be well padded and must immobilize the joints above and below the injury (Fig. 14.1). Where possible, splint with the limb in the anatomical position.

 Diagram of a field splint.
Fig. 14.1

Diagram of a field splint.

How did the injury occur? The mechanism is significant; for instance, twisting injuries tend to produce spiral fractures, while a fall onto the heels may produce fractures of the spine or base of skull.

Did the accident occur in a clean or contaminated environment? Consider IV antibiotics.

When did the injury happen?

Was the limb trapped or crushed? (Swelling and compartment syndrome are possible; see also graphic Crush injuries, p. 277.)

When was the last tetanus booster? This is very important for open fractures.

Is the patient allergic to anything?

Does the patient take any medication?

Look for contamination and foreign bodies.

Check for pulses and capillary refill time.

Examine carefully for signs of nerve damage: change in sensation, weakness, or paralysis.

In a wilderness situation, a fracture should be assumed until X-ray studies or clinical examination confirm otherwise.

Capillary refill time

To check for capillary refill press firmly over a fingernail or a bony prominence such as the sternum, forehead, or a malleolus for 5 s to produce blanching. When the pressure is released the colour should begin to return quickly (in <2 s). Slow filling indicates that the patient is extremely cold, shocked, or that the blood supply to the limb is interrupted.

Features of a fracture

Pain/tenderness.

Loss of function.

Swelling/bruising.

Deformity.

Crepitus.

A fracture is a soft tissue injury with an underlying break in the bone. Fractures are either open if the skin is broken or closed. They are comminuted if there are more than two fragments. Children’s bones may bend, leading to greenstick fractures. Complicated fractures involve damage to blood vessels, nerves, tendons, or organs.

Stop bleeding (may require a tourniquet or haemostatic agents).

Treat shock.

Monitor pulse, BP, and urine output.

Give adequate analgesia.

Take a digital photo of open fractures or significant soft tissue injuries.

Clean with antiseptic solution and cover exposed bone ends, for example, with saline-soaked gauze, and consider using antibiotics.

Consider reducing and then immobilize in an appropriate sling/splint.

Evacuate for X-ray and definitive fracture management.

A dislocation is an injury in which the normal relationships of a joint are disrupted. In some dislocations the bone end may be forced out of a socket (shoulder, hip, and elbow dislocations); in others the joint surfaces may simply be displaced (finger dislocations). Fractures, nerve, and blood vessel injuries may also be present with a dislocation.

Correction of dislocations can be technically difficult. Attempts to correct the deformity are justified in certain circumstances, particularly in remote areas. If the blood supply to the distal part of the limb is obstructed by a dislocation, reduction must be attempted. Steady, firm traction along the limb’s long axis may correct the deformity or at least relieve the obstruction temporarily. Reduction should be attempted as soon as possible because of increasing muscle spasm. After reduction, splint the limb as for a fracture.

(See Fig. 14.2.)

 Collar and cuff sling.
Fig. 14.2

Collar and cuff sling.

In the wilderness this may be improvised by passing a long sock around the patient’s neck and wrist of the affected side. This can then be secured using a cable tie or piece of cord. This uses the weight of the arm to apply slight traction to the upper arm and should be used for fractures of the humerus.

(See Fig. 14.3.)

 High arm sling.
Fig. 14.3

High arm sling.

This is mainly used to reduce hand swelling with hand injuries. Avoid excessive flexion of the elbow as this reduces venous drainage of the forearm.

(See Fig. 14.4.)

 Broad arm sling.
Fig. 14.4

Broad arm sling.

This is commonly used to support the weight of the arm and reduce movement in shoulder and clavicle injuries, dislocations/fractures of the elbow, forearm, and wrist. If used during evacuation of a walking casualty, a swathe around the chest placed on top of the broad arm sling further reduces movement. A broad arm sling may be improvised by pulling the lower edge of a jacket over the arm and then securing with a safety pin.

Arm slings and splints should be worn inside clothing to keep the hand and arm warm.

This is a common injury following a fall onto the outstretched arm. The clavicle is palpable along its length and there is often obvious deformity and localized tenderness. If the skin over the fracture is tented, then gentle traction with the arm out to the side will reduce the risk of developing an open fracture. Check movement, circulation, and sensation carefully. Treat with analgesia and a broad arm sling.

Injury to the acromio-clavicular joint commonly follows a fall onto the point of the shoulder. There is usually a characteristic step together with point tenderness at the joint. Treat with analgesia and a broad arm sling. X-rays will show the grade of injury, and the most severe may require surgical treatment.

The shoulder joint may be dislocated after violent injury (particularly forced abduction/external rotation) or after minimal injury in those with previous shoulder dislocations.

Most dislocations are anterior and are straightforward to diagnose as there is ‘squaring’ of the shoulder on the affected side and reduced movement, particularly abduction and forward flexion. The humeral head may be palpated antero-inferiorly to the glenoid fossa.

After any shoulder injury examine the area carefully for complications such as damage to the axillary nerve (loss of sensation over the insertion of deltoid—the ‘regimental badge area’). Axillary nerve damage merits expert assessment.

On an expedition it is reasonable to attempt reduction in the field, preferably with strong analgesia (± sedation such as midazolam). Reduce using the external rotation method or Spaso method. A clunk is often seen, felt or heard, and the shoulder’s normal contour is restored.

Sit the patient as upright as possible. Hold the patient’s elbow next to the trunk and, with the forearm mid-prone, slowly externally rotate the shoulder to 90°. The shoulder usually reduces at this point; if it does not, forward flex the upper arm slowly. An assistant may help to manipulate the humeral head into position.

Spaso Miljesic and Anne-Maree Kelly first reported the Spaso technique in 1998.1 This method is simple, needs minimal force, can be performed by a single operator, and is highly effective even in inexperienced hands. The Spaso technique is relatively atraumatic and countertraction is not required.

Lay the patient on their back and give sedation/analgesia as available. Grasp the affected arm around the wrist and slowly lift vertically to 90° shoulder flexion, applying gentle vertical traction as well as external rotation of the shoulder at the elbow. If difficulty is experienced, it may be helpful to use one hand to palpate the head of the humerus and gently push it to assist reduction, while maintaining traction with the other hand.

Lie the patient prone with the arm hanging down with a 5-kg weight attached to the wrist/hand. This method may take half an hour or more to achieve reduction.

Re-examine for axillary and radial nerve movement/sensation. Check pulses. Rest in a broad arm sling or collar and cuff. Evacuate for X-rays and orthopaedic follow-up/physiotherapy. Recurrent dislocations of the shoulder may not require an X-ray and can be managed with a broad arm sling and early mobilization.

This injury is rare and may follow electric shock or convulsions. It is easy to miss. It results from force applied to the anterior shoulder. The shoulder is internally rotated and there is marked loss of movement. Attempt reduction by applying traction and external rotation with the arm at 90° to the body. Manipulation under anaesthesia may be required.

The patient presents with the arm held above the head. Examine carefully for neurovascular deficits. Attempt reduction by applying traction along the abducted arm, then adduction. If it is not possible to reduce in the field, give strong analgesia, support the arm using padding with sleeping bags or similar, and evacuate for reduction under anaesthesia.

All conditions are caused by acute injury or soft tissue degenerative changes and may be provoked by unaccustomed activity as may occur on wilderness trips. The main symptom is of pain, often following lifting or a fall and onset may be sudden or gradual. Abduction and forward flexion in particular are restricted (unable to upturn a drinks can with arms outstretched in front of patient). There may be a painful arc of movement. Treatment with rest, NSAIDs, and a broad arm sling will usually reduce symptoms. Further assessment will be required if pain and restricted movements persist. Significant loss of ROM in the absence of pain may indicate a complete rotator cuff tear. Avoid prolonged periods of immobilization in a sling.

If crepitus is felt during manipulation of a shoulder dislocation, suspect an associated fracture and desist from further attempts at reduction in the field until the fracture has been identified or excluded by an X-ray. Support in a broad arm sling, give analgesia, and evacuate.

These are caused by a fall onto the outstretched arm or onto the elbow. Midshaft fractures can involve the radial nerve as it runs through the spiral groove and result in a wrist-drop.

Treatment for proximal injuries is with a collar and cuff sling. Midshaft injuries may benefit from a splint or broad arm sling and strapping arm against the body. Any involvement of neurovascular structures, particularly the radial nerve or brachial artery, should prompt urgent evacuation for definitive care.

This can be torn during lifting and may not require large forces. It is seen more commonly in elderly males. There is a characteristic bulge of biceps muscle above the elbow but often little pain; bruising may, however, be extensive. Surgical repair may be considered if the arm is not fully functional.

The elbow may be injured by a direct blow or transmitted forces such as a fall onto the outstretched hand. Full extension without pain makes the presence of fracture or serious injury very unlikely.

This requires considerable force and may be associated with fractures. The radius and ulna are usually dislocated posteriorly. Damage to the brachial artery or radial/ulnar/median nerves may occur. Attempted reduction is justified in a wilderness environment, particularly if evacuation to definitive care will take more than a few hours.

After suitable analgesia, apply steady traction to the limb by pulling at the wrist. The elbow is usually flexed at around 30°. Countertraction above the elbow with concurrent pushing of the protruding olecranon forwards is essential. Rest in a broad arm sling and evacuate for X-ray and further assessment/rehabilitation. If reduction proves impossible, place in a broad arm sling and give analgesia. Monitor radial pulse and assess for nerve damage.

Golfer’s elbow refers to inflammation around the common flexor origin at the elbow; tennis elbow involves the same process at the common extensor origin. Both conditions are caused by repetitive hand and wrist movements, particularly rowing and paddling on expeditions. On examination there is tenderness at the medial (golfer’s) or lateral (tennis) epicondyles of the humerus and pain on gripping.

Rest, anti-inflammatory drugs and, where possible, avoidance of the provoking activity and gentle stretching after 3–4 days.

The olecranon bursa may become inflamed and painful, sometimes after minor trauma. The lump over the elbow is fluctuant and may be very tender if it becomes infected.

This condition may take weeks to resolve but usually only requires rest in a broad arm sling and anti-inflammatory drugs. If there is evidence of spreading infection or fever then give antibiotics. Avoid aspiration in the field because of the danger of introducing infection.

Displaced fractures around the elbow may be associated with neurovascular injury, particularly the brachial artery. Check distal pulses and seek evidence of neurological deficit. Such fractures usually require orthopaedic assessment and often need internal fixation. With the forearm midprone, splint either in the position found, or with the elbow at 90°. Evacuate for X-rays and further management.

This injury can only be diagnosed with the help of radiographs but clinically is suggested by a history of a fall onto the outstretched hand and then pain and tenderness on palpating the radial head. Pronation/supination is often very painful. Support the forearm in a broad arm sling and evacuate for assessment.

These tend to be unstable fractures and may affect the radius and ulna at the same level, at different levels with spiral fractures, or involve a fracture of one bone with associated dislocation at one of the radio-ulnar joints. Check carefully for neurovascular compromise. Treat with analgesia, splinting, and evacuate for definitive care. If there is evidence of an open fracture, clean and dress the wound and give antibiotics.

These fractures are commonly caused by a fall onto the outstretched hand. There is often obvious deformity together with pain and swelling. Dorsal displacement of the distal fragment is most common—Colles’ fracture. For those familiar with a haematoma block, 6–8 mL lidocaine 2% can be injected into the fracture site before reduction—a haematoma block. All potential wrist fractures on an expedition should be supported in a suitably padded splint and rested in a broad arm sling. Imaging and definitive treatment will be required after evacuation from the field.

A fall onto the outstretched hand may lead to pain and swelling, together with difficulty gripping. If there is tenderness in the anatomical snuffbox, pain when ‘telescoping’ the thumb (pushing the straight thumb towards the wrist) or palpating over the scaphoid tubercle, a scaphoid injury must be considered. Avascular necrosis, non-union, and osteoarthritis may complicate scaphoid fractures. On an expedition, immobilize in a below-elbow splint, and evacuate for X-rays and follow up as long-term disability may result from undiagnosed scaphoid injury.

If the mechanism of injury makes a fracture unlikely and if findings on examination are non-specific, it is reasonable on an expedition to treat a tender wrist as for a sprain using a support bandage, anti-inflammatories, and early mobilization. If there is any uncertainty or if symptoms are not settling, it is safer to assume that there is a fracture and arrangements should be made to image the wrist in a suitable facility.

Inflammation of tendon sheaths may follow repetitive strain injuries. Characteristically there is pain on moving the wrist or thumb and a ‘creaking’ or ‘buzzing’ sensation may be felt over the affected tendons, usually on the dorsum of the wrist. Treatment consists of rest in a suitable splint for up to 3 weeks and anti-inflammatories.

Stop major bleeding using direct pressure and elevation. Consider the use of a temporary tourniquet (a BP cuff can be used).

Establish the exact mechanism of injury.

Remove rings early before swelling develops.

Hand injuries are very painful and adequate analgesia should be given promptly. After neurological assessment consider early use of local anaesthetic (LA), particularly ring blocks for finger injuries (see Fig. 14.6). Clean hand wounds carefully (the patient may be able to clean the surrounding area first).

Open fractures should be treated with careful cleaning and antibiotics.

Punching injuries with a break in the skin (usually at the knuckles) should be considered to be a bite wound (graphic Bites (animal and human), p. 276).

To avoid later disability, all hand injuries must be carefully assessed and managed.

 Local anaesthesia of hand.
Fig. 14.6

Local anaesthesia of hand.

Record whether the injury affects the patient’s dominant hand.

Compare both hands.

Look for swelling, deformity, redness, and wounds.

In the relaxed hand there is increasing flexion from the index to the little finger. A finger which is out of line should raise the possibility of a tendon or nerve injury. Rotation of the digit points to a fracture.

Ask the patient to make a fist and then fully extend fingers. Look for any obvious motor deficit and crossing of fingers.

Assess flexor digitorum profundus by holding the proximal interphalangeal joint (PIPJ) extended and asking the patient to flex the finger.

Assess flexor digitorum superficialis by holding the fingers not being assessed in extension. Ask the patient to flex the finger at the PIPJ.

Assess the lumbricals by flexing the fingers in turn at the metacarpophalangeal joints (MCPJs).

Finger extensors can be tested by placing the patient’s PIPJs level with a table edge then asking the patient to straighten the finger.

Test movements at the interphalangeal joints (IPJs), MCPJ, and carpometacarpal joints of the thumb.

Assess median nerve power by asking the patient to oppose the little finger and the thumb.

The ulnar nerve may be assessed by abducting or adducting the fingers.

Check sensation on each side of the digit (digital nerves), in the first web space dorsally (radial nerve), middle finger (median nerve), and little finger (ulnar nerve).

Fractures may be suggested by the mechanism of injury, swelling, bruising, deformity, and loss of normal function. In all cases, check for neurovascular deficit. If suspected, immobilize the hand in a boxing glove dressing (Fig. 14.5) and broad arm sling. Evacuate for imaging and further treatment.

 ‘Boxing glove’ dressing.
Fig. 14.5

‘Boxing glove’ dressing.

Dislocations can occur at the MCPJs or IPJs. After assessing for obvious fractures (small avulsion fractures cannot be diagnosed without imaging), check for any neurovascular damage. It is worth attempting reduction with in-line traction under LA (ring block). If successful, apply buddy strapping or use a boxing glove dressing (Fig. 14.5).

Attempt reduction, then immobilize in a suitable splint or ‘boxing glove’ (Fig. 14.5). Evacuate for definitive care as internal fixation may be required if there is an associated fracture.

These can occur at the metacarpophalangeal or interphalangeal joints. If the joint is grossly unstable when gently stressing the collateral ligaments, surgical treatment may be required. For most ligament injuries, rest with neighbour strapping will allow healing to occur. Warn patients that swelling may take weeks to settle. This occurs in particular with injuries to the volar plate at the PIPJs. If the mechanism of injury involves a forced abduction at the thumb MCPJ (gamekeeper's/skier's thumb), ensure that laxity of the ulnar collateral ligament is tested for. If there is no end point on stressing, surgical repair will be required.

Small cuts or lacerations may damage tendons and these injuries may easily be missed unless hand injuries are carefully assessed. (Beware of any injuries caused by broken glass—evacuation for X-ray is recommended.)

Loss of function is the only reliable sign that a tendon has been damaged, but pain out of proportion to the injury should suggest damage to underlying structures. Ensure that you examine the wound with the fingers moving through their range of movement as tendons move with the finger so the injury may not initially be visible.

This injury is usually obvious. Partial tendon ruptures are easily overlooked—explore wounds under LA or evacuate for full assessment in a bloodless field.

Flexor digitorum superficialis flexes the PIPJ. Flexor digitorum profundus flexes the distal interphalangeal joint (DIPJ).

To test these tendons see graphic Examination of the hand, p. 430.

Tendon injuries should be repaired in a suitably equipped hospital, not in the field, because of the danger of infection of the flexor sheath that can cause long-term disability.

On an expedition, fingers can be injured in many ways; they may be crushed in vehicle doors or under heavy weights or during the use of tools/machinery. After release, the digits should be carefully examined (under LA if possible), cleaned, and elevated. Open fractures should be given an antibiotic such as co-amoxiclav and evacuated for X-ray and further management. Always remember to remove rings.

Rupture of the central slip of the extensor tendon to the distal phalanx results in loss of extension at the DIPJ. This may be associated with an avulsion fracture of the base of the distal phalanx. On an expedition, splint the DIPJ in extension for 6 weeks and arrange for orthopaedic review on return. Do not remove the splint as movement will disrupt the healing tendon.

Provided the bone is not exposed and the area of skin loss is <1 cm2 it may be possible to treat these injuries in the field. After assessment, clean and dress (using Vaseline® gauze or similar). Re-examine every 2 days. If the wound is not healing, evacuate for imaging and possible terminalization of bone or skin grafting.

Finger entrapment or crushing injuries may result in partial or complete avulsion of a nail. The finger should be anaesthetized (Fig. 14.6) and cleaned. The patient may be able to help to clean the finger by immersing in warm, clean water. If the nail is displaced it should be removed under LA, any defect should be repaired, the nail should be replaced for protection and the finger dressed and splinted.

Crush injuries to the nail may result in bleeding under the finger nail. The pressure results in throbbing pain which may be relieved by heating a paper clip to red heat and burning a hole in the centre of the nail, using minimal pressure. On an expedition a Leatherman® multi-tool can be used to hold the heated wire paper clip. It may be necessary to reheat the wire on several occasions. Alternatively, a 21 G hypodermic needle can be used to drill a hole in the nail by gentle twisting motion. Once the blood has drained the pain is significantly reduced.

Splinters of metal or wood under the finger nail are common. A ring block may allow removal with splinter forceps or trimming of part of the nail to allow removal.

See graphic Paronychia, p. 297; Fig. 9.6.

Many fractures to the lower limbs are high-energy injuries and are associated with other injuries:

Falls from heights may lead to heel, femoral, and pelvic fracture, but 10% have associated spinal injury—this may be masked by pain at the obviously fractured leg.

Fall from heights >2 m and lower limb fracture = spinal injury until excluded.

Dashboard injury—knee injury in seated passenger during road traffic collision (RTC) associated with femoral fracture and hip dislocation/acetabular fracture.

Bony injuries to this part of the body are always serious and usually associated with major blood loss. Blood loss for closed lower limb fractures as a proportion of the total circulatory volume are shown in Box 14.1. Blood loss following open fractures may be much greater.

Box 14.1
Blood loss

Pelvis: 20–80%.

Femur: 20–40%.

Tibia: 10–25%.

These follow high-energy trauma such as falls and RTC. Considerable force is required to disrupt the pelvis, therefore these fractures are frequently associated with other major thoracic/spinal/abdominal or skeletal injury. Major vessels and pelvic organs lie adjacent to pelvic bones, and these may also be damaged. Mortality rates are ~10–20%.

Lateral compression—deforming force from side-to-side, e.g. vehicle rollover.

Anterior–posterior compression— this may give rise to the ‘open book’ fracture where the pelvis opens up; e.g. as a result of a rider hitting the petrol tank of a motorcycle.

Vertical sheer—usually the result of a fall from a height onto one leg, displacing the hemi-pelvis vertically.

If a pelvic injury is suspected by mechanism, pain, or hypovolaemic shock (see graphic Hypovolaemic shock, p. 234), treat as a fracture. Do not perform unnecessary examination that will exacerbate bleeding and do not log roll the patient unless absolutely necessary.

Treatment is aimed at stabilizing the pelvis and reducing further haemorrhage/visceral damage during onward transfer of the patient.

Control lower limbs—splint legs together at knees and ankles. Flex the knees about 20° and pad any bony prominences.

Splint pelvis—wrap a folded sheet firmly around the pelvis (upper border of sheet level with anterior superior iliac spines) and tie at the front or use a SAM Pelvic Sling® if available.

Evacuate urgently and move as little as possible.

The elderly and those with osteoporotic bones may fracture the neck of the femur with relatively minor trauma; however, the same fracture in younger patients indicates high-energy trauma such as a climbing fall.

Often there is a history of a fall onto the lateral aspect of the hip. Pain may radiate to the knee and the affected leg may be shortened and externally rotated. Give analgesia; look for and treat shock. A traction splint may improve comfort during evacuation (see graphic Femoral fracture, p. 437). If experienced, consider a fascia iliaca nerve block (see graphic Fascia iliaca block, p. 594).

These usually follow a fall from a height or dashboard injury in RTCs. The hip joint usually dislocates posteriorly with or without fracture of the acetabulum. Travellers with hip replacements are at greater risk of dislocation with minimal trauma.

There is pain and deformity of the leg, which is shortened and rotated (internally if posterior dislocation). Assess neurological status and distal pulses—hip dislocations may be associated with injury to the femoral or sciatic nerve. If immediate evacuation is possible then splint legs in position and give strong analgesia. If >6 h before evacuation to hospital, then blood supply to the femoral head may become compromised and an attempt at reduction is justified but will require adequate analgesia ± sedation.

In posterior dislocation, the assistant places hands as countertraction on the pelvis (anterior superior iliac spine). Flex the hip to 90° with the knee bent and then apply vertical traction with internal rotation. As the hip relocates externally rotate, extend the hip, and continue longitudinal traction to the lower leg. Wrapping a bandage around the lower leg and foot may allow application of 2.5–5 kg skin traction to be maintained. Check neurovascular status post reduction—relocation may be prevented by entrapment of the sciatic nerve around the femoral neck. The hip may re-dislocate owing to an unstable acetabular fracture. Repeated attempts at reduction are not justified—evacuation is required ASAP for operative intervention.

A great deal of force is required to fracture the femur—usually following a fall from a height or RTC. It can be associated with other head, spinal, chest, or abdominal/pelvic trauma.

There is pain and deformity of the thigh, together with swelling and fracture crepitus. Assess neurological status and distal pulses. Anticipate and whenever possible treat hypovolaemia (the patient may lose 20% of blood volume rapidly even in closed injury). Splint legs in position (splint to un-injured leg); apply traction through a splint if available to maintain alignment and to reduce bleeding into the thigh compartment. Cover any open wounds with sterile dressings, e.g. saline-soaked gauze.

Strongly consider a fascia iliaca block if suitably experienced (see graphic Fascia iliaca block, p. 594).

Knee injuries on expeditions are relatively common. In all cases take a careful history which may give clues to the diagnosis. Ask about previous knee problems such as swelling, clicking, locking, or ‘giving way’.

Examination of the injured knee:

Look for bruising, swelling, redness, deformity, and compare with the uninjured side.

Feel for an effusion, warmth, or crepitus. Identify any tender areas—joint line or origin/insertion of collateral ligaments.

Observe straight leg raising (which assesses the extensor mechanism, graphic Knee extensor mechanism injuries, p. 442).

Assess movement—extension (0°), flexion (135°).

Palpate along the medial and lateral joint lines, and over the fibular head for tenderness.

Test the joint stability—in 30° flexion support the lower leg and apply valgus stress (medial collateral) and in extension a varus stress (lateral collateral) at the knee. With the knee flexed at 90°, place the thumbs on the tibial tubercle and rest index fingers behind the knee. With the hamstrings relaxed, gently draw the lower leg forward, looking for any abnormal shift (anterior draw test).

McMurray’s test for meniscal injury—place thumb and index finger on medial and lateral joint lines, flex the knee, and externally rotate the foot followed by abduction and extension of the knee. Pain and a click suggest a medial meniscal injury. In the acute knee McMurray’s test can be difficult and medial or lateral joint line tenderness can be as useful in identifying meniscus injuries.

This is relatively common and refers to bleeding into a joint. Rapid tense swelling develops in 1–2 h. Haemarthrosis may be spontaneous (coagulation disorders and rare vascular tumours) or traumatic (80% after anterior cruciate ligament injury, 10% patella dislocation, 10% meniscal injuries/capsular injuries).

RICE, analgesia, and evacuate for further assessment.

There will be good range of movement at the knee. Inflammation of the fluid-filled bursa in front of or below the patella may result from unaccustomed, frequent minor trauma such as kneeling. Rest and NSAIDs usually relieve symptoms. If there are features of spreading cellulitis and a fever, antibiotics should be given (e.g. co-amoxiclav).

Sudden knee flexion or a direct blow may result in a fracture of the patella. There is usually pain, swelling (sometimes from a haemarthrosis), and inability to straight leg raise. If suspected, splint the leg almost straight (with 5° of flexion at the knee) and evacuate for imaging and definitive treatment.

Suspected fractures around the knee should be treated with adequate analgesia and splint, as for patellar fracture. Avoid traction splints if there is a possible fracture in the supracondylar region of the distal femur. Traction may displace the distal part of the fracture posteriorly and damage the popliteal artery.

This is not uncommon and may be a recurrent problem (more common in females). The patella dislocates laterally and, typically, the patient’s knee is held flexed with obvious displacement of the patella. Give analgesia and reduce the patella by pressing with the thumbs on the lateral aspect of the patella as the knee is straightened. Once reduced it may be possible to rehabilitate this injury in the field without evacuation.

This is a rare injury and huge forces are required to produce disruption of the knee ligaments. There is a high likelihood of nerve and blood vessel damage—check distal pulses and sensation carefully. Treat with strong analgesia and immobilize as for patellar fracture before evacuation. Keep monitoring foot pulses, as popliteal artery injury may not be apparent initially. If there are signs of vascular compromise, evacuate urgently for vascular surgery.

The medial collateral ligament runs from the medial epicondyle of the femur to 4 cm distal to the knee joint on the medial aspect of the tibia. An isolated rupture usually results from a direct blow to the lateral aspect of the knee in slight flexion. If there is a rotational component, such as a fall when skiing, there may also be injury to the cruciate ligament.

On examination there is tenderness over the medial collateral ligament ± knee swelling. Test valgus stability with knee flexed 20–30º (in full extension the cruciate ligaments stabilize the knee).

Mild–moderate (<10 mm opening of joint)—rest, ice, compression, and then early increase range of motion/strengthening with physiotherapy.

Severe (>10 mm opening of joint)—may require hinged brace for 3–6 weeks after initial management and so will benefit from evacuation for definitive care.

The lateral collateral ligament runs from the lateral epicondyle of the femur to the head of the fibula. Isolated injuries are rare but are more usually associated with injury to all the lateral capsular ligamentous structures. This may result in marked instability.

Tenderness ± knee swelling. Test varus stability. An isolated lateral collateral ligament injury may be treated as for medial collateral ligament, but more common complex injury may require evacuation for surgery.

Anterior cruciate ligament injuries account for 50% of documented knee ligament injuries. Posterior cruciate ligament injury is rare (10% knee ligament injuries). Usually, injuries are caused by non-contact twisting injury, occasionally following hyper-extension. There is pain and difficulty weight-bearing. Swelling usually develops immediately with an ACL rupture. A PCL rupture will have no swelling and a dull posterior knee ache.

There is an acute haemarthrosis following typical history. Examination is often difficult during the acute phase due to pain and swelling. A subjective description of a ‘popping’ sensation is often noted.

The Lachman test will usually be positive in an ACL rupture—grip the tibia at 30° flexion and pull it anteriorly over the distal femur. There will be no firm end point. There may also be anterior draw and pivot shift. If suspected, evacuate for X-rays and further imaging (MRI) or arthroscopy. Younger active individuals may have continuing joint instability and may require reconstruction of the cruciate ligament. The PCL will have a positive posterior draw at 90°.

The menisci act as stabilizers for the knee and distribute forces across the articular surfaces. There is usually a history of an axial load with a twisting injury to the knee. ‘Degenerative’ tears may occur in patients >35 years with very little history of injury.

Patients may complain of the knee ‘locking’ or ‘giving way’. Pain and intermittent swelling may occur. Squatting particularly may aggravate posterior horn tears.

Acute tears may cause gradual swelling of the knee over 4–6 h and occasionally haemarthrosis. Degenerative tears may settle with physiotherapy.

Knee arthroscopy is often required if the knee is symptomatic. If the knee is acutely locked, from a loose body, do not attempt to unlock the knee (painful and usually futile). Splint in a comfortable position and evacuate for definitive care.

Disruption to any one of these may prevent straight leg raising or active extension of the knee. (NB Tense knee effusion and pain may also prevent straight leg raise without disruption of extensor mechanism.)

80% occur in individuals >40 years. Normally a gap is palpable close to the superior pole of the patella (defect at insertion of vastus medialis).

Usually occurs in those >40 years. Tender inferior pole patella.

Usually occur as result of direct trauma.

Manage with rest, support, and splinting in extension. Tendons usually require operative repair if the extensor mechanism is disrupted. Fractures often require reduction and fixation if displaced.

This can occur as a result of a fall, RTC, or twisting injury (particularly when skiing). There is often an associated fibula fracture. Because there is little soft tissue cover, tibial fractures are commonly open.

There is a significant risk of compartment syndrome whether an open or closed injury (see graphic Crush injuries, p. 277; graphic Compartment syndrome, p. 443).

Splint legs in position (splint to the other uninjured leg); apply traction through a splint, if available, to maintain alignment. Cover open wounds with a sterile dressing (take a photo first) and give antibiotics during evacuation, e.g. co-amoxiclav.

This refers to increased soft tissue pressure within an enclosed soft tissue compartment which can lead to devastating muscle necrosis and nerve damage.

The soft tissues swell but the surrounding envelope does not allow expansion, causing pressure to rise above the capillary pressure. It usually follows fractures or crush injuries (particularly to the lower leg, thigh, forearm, and foot/hand), and is most common following closed fractures to the tibial shaft. Occasionally it occurs as a chronic exercise-induced condition (‘shin splints’).

Try to prevent occurrence in the field with elevation of the affected limb above the level of the heart. The soft tissue compartment looks swollen and tense. The most reliable sign is pain on passive stretching of a muscle group within the compartment, e.g. great toe or fingers. Remember the 5Ps—Pain, Pallor, Paraesthesia, Paralysis, and Pulseless, reflecting tissue ischaemia. Pain ‘out of proportion to the injury’ and local tenderness are the earliest features and are difficult to control with even opiate analgesics. Loss of pulse is a very late sign.

Acute compartment syndrome is an emergency requiring urgent evacuation and surgical decompression.

This refers to micro-tears in the Achilles tendon. It usually affects those aged 35–55 years and follows unaccustomed activity such as running, trekking, jumping or direct trauma to the tendon.

This occurs in two areas:

Non-insertional—4–8 cm proximal to Achilles insertion into calcaneum. It is probably a result of reduced blood supply and a point of high tension in the tendon. A tender fusiform swelling develops.

Insertional—occurs at the insertion of the Achilles into the calcaneum. A hardened bony lump develops, causing painful rubbing and difficulty with shoes/boots. A bony prominence on the posterior–superior aspect of the calcaneus (Haglund’s spur deformity) may be present along with an inflamed bursa (retrocalcaneal bursitis).

Conservative measures improve symptoms in 90%. In the acute stage, RICE and regular NSAIDs are required. Modify activity to reduce strain on heels.

After the acute phase has settled consider Achilles stretching exercises (eccentric loading) and removal of heel tabs in training shoes. A heel-raise to reduce stretching of the tendons can also be considered. There is a small risk of complete rupture.

There is a relatively poor vascular supply and high tension ~4–8 cm proximal to the insertion into the calcaneum. Age is usually 35–55 years. The injury occurs during sudden contraction during ‘push-off’ or an injury with forced dorsiflexion of the ankle.

The patient complains of sudden pain and weakness of plantar flexion at the ankle. A ‘gun shot’ may be heard as the tendon ruptures. On examination there is tenderness and a palpable boggy gap in the tendon.

Simmonds’ test-positive—lay the patient prone with foot over end of bed. Gently squeezing the calf causes plantar flexion at ankle in the unaffected side but no or minimal plantar flexion with rupture of Achilles tendon.

Initial management is RICE and splint ankle in equinus position (foot plantar flexed), non-weight-bearing. Evacuate for expert assessment and management. Currently there is considerable debate regarding non-operative treatment in equinus cast for 9 weeks (slow recovery and possibly higher re-rupture rate) versus operative repair (wound problems and sural nerve injuries).

Shin splints—pain over the anterior shin muscles following running may be treated with rest and NSAIDs.

Gastrocnemius/soleus muscle tear.

Stress fracture of the tibia (see graphic Lower limb stress fractures, p. 446).

DVT.

Cellulitis (see graphic Cellulitis, p. 278).

These commonly occur during a forced inversion injury of the hindfoot. Generally the lateral ligaments are injured—anterior talo-fibular ligament and calcaneo-fibular ligament. Bruising and swelling may be severe, occurring within hours. Tenderness is usually maximal laterally.

A fracture may be difficult to exclude clinically; however, the Ottawa rules suggest that X-rays should be arranged if the patient is unable to weight-bear (four steps) or if there is tenderness behind the medial or lateral malleolus, over the navicular (proximal to base of first metatarsal), or at the base of the fifth metatarsal. Palpate the fibular head as ankle injuries can cause fracture of the fibular head.

RICE and analgesics. Strapping may be beneficial and supporting the ankle initially whilst allowing weight-bearing as tolerated. Active exercises should begin immediately, as pain allows, to regain full movement. Persistent or worsening pain on weight bearing after 1 week may require further review.

Normally the talus sits in the mortise of the tibia and fibula; twisting may rupture the ligaments or fracture the malleoli. This occurs when the foot is anchored on the ground and the momentum of the body continues forwards. There is swelling, pain, and bruising, with deformity and tenderness along the bony landmarks of the medial and lateral malleoli.

If the ankle is obviously dislocated, this may cause pressure necrosis on the soft tissues and neurovascular compromise. Reduction by traction and relocation is indicated urgently (with suitable analgesia). After reduction, check pulses and sensation then immobilize in a suitable splint. Treat with RICE. X-rays are needed to confirm the nature of the fracture. Evacuate for definitive treatment; many displaced ankle fractures will require operative fixation.

Foot injuries can be devastating on trekking expeditions as the victim may require stretcher evacuation. The feet are particularly vulnerable when inadequately protected by lightweight footwear in the tropics or jungle.

Falls from a height onto the feet can result in fractures around the heel. These may be bilateral and are associated with other significant injuries such as to the spine, pelvis, hips, or knees.

Give strong analgesia, elevate, and immobilize in a splint. Early definitive treatment is necessary to prevent long-term disability.

Heavy weights such as vehicle wheels or large machinery may break multiple metatarsal bones. Ankle inversion may lead to a small avulsion fracture at the base of the fifth metatarsal.

Check for pulses and evacuate for injury assessment and definitive care.

These most commonly affect the metatarsals, but can occasionally occur in the tibia, the calcaneum, or talus. They follow repeated trauma such as occurs in over-training. There is an underlying biomechanical problem that may be resolved with a change in shoes/boots.

Metatarsals—there is gradual onset of pain ± swelling of the forefoot without specific trauma. Tenderness is specifically along the bone (usually second metatarsal neck). Early X-rays are often normal but may show callus formation at 3–6 weeks.

Tibia—shin-splint pain on exercise. Pain may settle after a period of rest. May have tenderness along the medial border of the tibia.

Calcaneum — exertional non-specific heel pain. Squeezing heel causes pain. (NB Plantar fasciitis may cause pain under the heel when walking, which is normally worse for the first few steps after rest, but there is tenderness specifically at the insertion of the plantar fascia under the heel.) X-rays are normal.

In all cases give analgesia and remove the cause, often necessitating a reduction in training/exercise. Reduce weight-bearing with crutches until comfortable and then start mobilization, as tolerated.

These rarely require any more than ‘buddy strapping’ to relieve pain for 2–3 weeks. If there is obvious deformity, this may be reduced under ring block (inject 1 mL plain lidocaine 1% on each side of the base of the digit).

These are painful, difficult to manage, and prone to infection. To anaesthetise the area, consider topical LA such as Ametop®, or regional anaesthesia by performing a nerve block of the posterior tibial nerve just posterior to the artery where it runs behind the medial malleolus. This will anaesthetize the majority of the sole of the foot. If the wound is on the lateral sole, consider a nerve block of the sural nerve as it runs behind the lateral malleolus. The tough skin of the sole makes local infiltration of LA painful and ineffective. Remove any foreign bodies and close the wound with glue or Steri-Strips™. There is a significant risk of infection—puncture wounds should be treated with antibiotics prophylactically. Ensure tetanus immunization is up to date.

This is inflammation of the connective tissue that forms the sole of the foot. There may be pain over the heel, under the arch of the foot forward to the metatarsal heads. It commonly occurs in individuals not accustomed to exercise who suddenly increase exercise intensity. Pain is common for the first few steps of the day or on dorsiflexion of the foot. The condition can be eased with calf stretching exercises and orthotics or cushioned footwear. It can last for up to 2 years.

(See also Chapter 7 and Chapter 14.)

Spinal cord injuries are relatively rare. However, it is vital that the possibility of spinal injuries is considered in all trauma patients especially:

High-speed injuries.

Patients with multiple injuries.

Falls or those who have been hit by a falling object such as a rock.

Head-injured and unresponsive patients.

The implications of missing one of these can be life-changing or life-threatening for the patient. Manipulation or inadequate management/ immobilization of the spinal-injured patient can cause additional neurological damage and worsen outcome.

Injuries most frequently occur at junctions of mobile and fixed sections of the spine (C6,7/T1, T12/L1). In patients with multiple injuries who have a spinal injury, over half will have a cervical spinal injury. About 10–15% of patients with one spinal fracture will be found to have another.

The aim of spinal management is to prevent any secondary injury to the spinal cord. If a casualty with a spinal injury is moved carefully, they are unlikely to come to further harm but do follow these precautions:

Ensure that the airway is open. Use the jaw-thrust manoeuvre rather than head tilt and chin lift, which may create further cervical spine injury.

If necessary, move the head into a neutral alignment (Fig. 14.7). Stabilize the head and neck manually. If available, use a semi-rigid neck collar such as Stifnek select™ together with blocks and tape—in the wilderness socks full of sand or soil can be used.

Assess breathing and look for and treat any life-threatening chest injury (graphic Breathing, p. 190). Give oxygen if available to keep sats >94%. Adequate oxygenation and tissue perfusion must be maintained as the spinal cord is very sensitive to hypoxia and hypotension.

Identify any signs of shock and treat cautiously with IV fluids. Ensure an adequate BP (systolic 90 mmHg) and pulse rate. If the pulse rate falls below 45 bpm consider atropine 600 micrograms IV as minimal stimulation may cause asystole owing to unopposed vagal response.

Rapidly assess the conscious level with AVPU scale or GCS, look at the pupils, and ask the patient if they can move and feel their fingers and toes.

Only log roll the patient if you suspect a penetrating injury to the back or wounds that require attention. There is little benefit in palpating the spine or performing a rectal examination. Unnecessary log rolling may exacerbate a spinal injury but more importantly an unstable pelvic injury.

If the patient must be moved, do so with caution but ensure that pressure areas are padded and if there is a spinal injury causing bowel and/or bladder dysfunction, ensure attention is paid to keeping the skin clean and dry.

 Manual immobilization of the neck.
Fig. 14.7

Manual immobilization of the neck.

Not all patients who are involved in trauma need to have full spinal precautions maintained. Box 14.2 indicates when a spinal injury can safely be excluded clinically.

Box 14.2
Excluding spinal injury (‘clearing the spine’)

No midline cervical tenderness.

No altered level of alertness.

No evidence of intoxication.

No neurological abnormality.

No distracting injury.

If the patient has any of these signs then imaging of the neck is required before cervical spinal immobilization can be removed.

If it is necessary to move the patient, they should be moved carefully, keeping the spine in alignment throughout and should be kept horizontal if at all possible. This is because blood vessels below the level of a spinal cord injury may have lost their spinal reflexes and so cannot contract in response to hypotension. This can cause a precipitous drop in BP and hence further damage to the spinal cord.

Signs of a spinal injury

The patient may complain of:

Neck or back pain. This may be masked by another more painful injury.

Loss of movement and/or sensation in the limbs.

Sensation of burning/electric shock in the trunk or limbs.

On examination the patient may have swelling or midline tenderness over the spinous processes.

In the unconscious patient a serious spinal injury may be indicated by:

Hypotension with bradycardia (neurogenic shock).

The skin may be warm below the level of the lesion.

Diaphragmatic breathing.

Flaccid tone.

Priapism (involuntary erection of the penis).

Loss of sphincter control.

Back pain is a common complaint and on expeditions may be provoked by unaccustomed activity or injuries such as lifting awkwardly, falls, or twisting injury. 80% will resolve within 2–8 weeks. Low back pain is very common. Sort into:

Mechanical back pain (most prevalent).

Nerve root pain—only concerning if progressive or persistent.

Serious spinal pathology—will need definitive care.

Traumatic.

Infectious.

Visceral (~2%).

Suspected cord compression—needs immediate evacuation.

Consider other systemic disease, such as back pain associated with weight loss, fever/rigors, cough/haemoptysis.

Recent back trauma—note the mechanism of any injury.

Characterize the pain, noting particularly leg symptoms and aggravating and relieving factors.

Ask if there is any disturbance of bladder/bowel function.

Ask about previous back injuries or surgery.

Ask about history of cardiovascular disease, e.g. stroke.

Presence of red flag signs.

Red flag signs

Uncontrolled pain, worse at night.

Fever and/or unexplained weight loss.

Loss of bladder or bowel control.

History of carcinoma—particularly thyroid, breast, lung, prostate, kidney.

Ill health or presence of other medical illness.

Significant motor weakness or sensory loss.

Previous osteoporotic fractures.

Disturbed gait, saddle anaesthesia.

Age of onset <20 or >55 years.

‘Unwell’ patient—immediately assess ABCs and check for presence of pulsatile expansile abdominal mass and presence/absence of femoral pulses (abdominal aortic aneurysm often presents with back pain).

‘Well’ patient—look for signs of weight loss, scoliosis, and muscle spasm. Watch the patient walk, looking for limping or abnormal posture. Assess spinal movements and note any significant loss. With the patient supine, palpate for tenderness over lumbar spine and sacrum, ribs, and renal angles. Look for muscle wasting in the legs. Perform on both legs:

Straight leg raise—note angle at which patient detects pain (lumbar nerve root irritation). Normal 70° but compare with other side.

Check for perineal and perianal sensation. Consider rectal examination to check anal tone.

See Table 14.1.

Table 14.1
Neurological examination
SensationMotorReflex

L3/4

Medial lower leg

Quadriceps

Knee jerk

L5

Lateral lower leg

Extensor hallucis longus

Hamstring jerk

S1

Lateral foot and little toe

Foot plantar flexors

Ankle jerk

SensationMotorReflex

L3/4

Medial lower leg

Quadriceps

Knee jerk

L5

Lateral lower leg

Extensor hallucis longus

Hamstring jerk

S1

Lateral foot and little toe

Foot plantar flexors

Ankle jerk

If red flag sign is found, evacuate for specialist investigation of the cause of the back pain.

In the absence of red flag signs, even with the presence of nerve root pain, conservative treatment should be effective.

Manage symptoms with:

Reassurance and education that most back pains completely resolve in 2–8 weeks.

Simple analgesics and NSAIDs, and a short course of a low dose of benzodiazepines (2–5 mg diazepam three times a day for 2 days). Consider opioids for a limited period if needed.

Use heat not ice.

Avoid bed rest; encourage gentle, normal movements.

Consider physiotherapy or manipulation if available.

Always keep the diagnosis under review if symptoms change.

Most soft tissue injuries will benefit from a short period of rest/ protection (splinting) followed by early mobilization to prevent stiffness and to regain full movements.

Traditionally RICE forms the basis for simple soft tissue injury rehabilitation.

Rest (for 24–48 h) and ice, if available, applied to the injured part help to reduce swelling and pain. Ice should not be applied directly to the skin but should be wrapped in a damp cloth and applied intermittently for 15 min at a time. If ice is not available, cold packs would be a suitable alternative or even placing the injured limb in cold water.

A crepe or tubigrip bandage may help to support the injury and will remind the patient about the injury. If the patient must continue to bear weight, compression and ice are beneficial. If complete rest is possible, omit compression and instead elevate and use ice.

Hand and foot injuries benefit from early elevation to reduce discomfort and swelling. However, poorly applied elbow or knee compression may be harmful if venous return is affected (risk of DVT).

Gentle range of motion exercises should start as soon as possible. Aim for graded exercises which move joints slightly more each day. Avoid excessive stretching and try to normalize movement as early as possible within the limits of pain. Always provide adequate analgesia.

1–3 days—rest, ice, elevate.

3–14 days—perform regular exercises building up activity, avoiding excessive discomfort or strain.

14 days onwards—injury requires less protection. Concentrate on getting the injured part back to full fitness with exercises.

By 8 weeks all usual activities should have been resumed.

Taping or strapping is simply the application of adhesive tape to provide extrinsic stability and/or to offload weakened structures. Its proprioceptive qualities play a significant part in protection and rehabilitation. The principal aim of applying tape is to prevent disability and thus improve otherwise impaired physical function. Techniques can be used for a multitude of musculoskeletal conditions, either following tissue injury or as a preventative measure when a history or risk of injury is present. (See Fig 14.8.)

 Ankle strapping.
Fig. 14.8

Ankle strapping.

The most commonly used types of taping material are the adhesive elastic type (such as Elastoplast®) and zinc oxide tape (a non-stretch, highly adhesive tape). The indications for each type depend on the aim of the technique being used, but often both types will be used concurrently; e.g. elastic adhesive tape is ideal for providing anchor strips as these are often circumferential strips of tape and must stretch to allow contraction of muscle from these anchor strips. Zinc oxide tape can be used to act as a non-stretchy stabilizer to prevent particular joint movements. Zinc oxide tape requires changing every day as after stretching it over a day, it will be no longer supportive.

In a number of techniques a pre-taping under-wrap can be used to prevent direct contact of the tape with the skin. However, it is the traction of the skin–tape interface that is thought to give the techniques their effectiveness and therefore the use of under-wrap can be questioned.

Prior to undertaking any taping technique, patients should have undergone a comprehensive assessment of the injury with particular reference to mechanism of injury, precise direction of any resultant instability/ weakness, and the phase of healing that is likely to be ongoing. These factors will lead to a clinical decision being made regarding the selection of taping material as well as the technique that is to be applied. Patients should also be questioned as to whether any known allergy to the taping materials is known.

Adequate preparation of the contact area is vital to ensure ease of technique and a good skin–tape interface. The following can be used as a guide:

The area to be taped should be clean and dry.

Hairy areas should be shaved.

Areas of broken skin should be covered with a non-adherent dressing such as Telfa® or Melonin®.

The patient should be positioned to allow the therapist access to the taped area without having to reposition throughout the technique.

Once the tape is applied, simple checks to observe circulation to the area should be performed.

Acutely injured joints are at greater risk of further injury owing to the loss of proprioceptive input caused by painful stimuli. The use of taping as an adjunct to improving joint stability extrinsically is commonly used in the sports setting and is aimed at restoring function at an early stage whilst protecting structures from further injury. Adhesive tape has been found to provide compression to acute ankle sprains for up to 10 days post injury.2 It should be noted, though, that applying tape for such prolonged periods can be counterproductive as it will limit venous flow from the injured area and thus prevent resolution of swelling.

In the presence of swelling around an acutely injured joint, it is often wise to ensure that the circumference of the joint and the surrounding muscular tissues are not completely covered. This allows for further swelling to take place without causing any vascular compromise.

In acute muscle strain, longitudinal taping crossing both the origin and insertion of the muscle can prevent further injury and reduce disability by limiting the muscle’s potential to lengthen. This technique, known as physiological taping, can also be used during rehabilitation to offload the muscle at the point of stretch and, through the elastic quality of the tape, it can assist muscle contraction.

The most common use of musculoskeletal taping is as a preventative measure when returning athletes to sporting activity. Taping itself has been compared to functional sports bracing as a means of providing effective joint stability. No difference was found between the overall effectiveness and practicality of taping versus a laced ankle support across a number of sporting activities, but taping has the advantage of being adaptable to the individual athlete or expedition team member.

MacDonald R (

2004
).
Taping Techniques, Principles and Practice
, 2nd edn. London: Butterworth–Heinemann.

Notes
1

Miljesic S, Kelly AM (1998). Reduction of anterior dislocation of the shoulder. Spaso technique. Emerg Med, 10, 173–5.reference

2

Capasso G, Maffuli N, Testa V. (1989). Ankle taping: support given by different materials. Br J Sports Med, 23, 239–40.reference

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